Treatment of stage 3 chronic kidney disease is a worldwide health problem

Treatment of stage 3 chronic kidney disease is a worldwide health problem. The disease is most often progressive of nature with a high impact on patients and society. It is increasingly recognized that Treatment of stage 3 chronic kidney disease  can be detected in the early stages and should be managed as early as possible. Treatment of the cause, but in particular control of the main risk markers, such as high blood pressure, glucose and albuminuria, has been instrumental in delaying the progression to end-stage renal disease (ESRD).

There are many causes of chronic kidney disease, with diabetes and hypertension (high blood pressure) being two of the most common. Some types of kidney disease can be treated and cured, while others eventually lead to loss of kidney function and the need for dialysis.

However, even if the actual cause of a Treatment of stage 3 chronic kidney disease - such as diabetes or hypertension - cannot be removed, there are things that can be done to minimize or slow the damage done by the kidney disease, and to postpone the need for dialysis for as long as possible.

The two principle outcomes Treatment of stage 3 chronic kidney disease are progressive loss of renal function, and the development and progression of cardiovascular disease (CVD) [1]. The aim of this Background Paper is to discuss the evidence for treatments to slow the progression of both CKD and the attendant CVD, and to discuss treatments for the metabolic consequences of Treatment of stage 3 chronic kidney disease. The evidence was gathered using a Medline search of primarily meta-analyses where available, as well as randomized controlled trials (RCTs) from 1996–2006.

Although the rate of progression of CKD is related to some non-modifiable characteristics such as race, baseline renal function, male gender and increased age, there are a number of modifiable characteristics. It is important to note, however, that the three most widely studied interventions, blood pressure (BP), proteinuria and drugs to reduce both, are inextricably linked in their effects on glomerular filtration rate (GFR).

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